ADHD is associated with altered dopamine regulation across brain systems involved in:
1. The “Executive” Part of the Brain
The front part of the brain, called the prefrontal cortex, acts like a management system.
It helps you:
2. The Brain’s “Brake System”
A key difference in ADHD is in inhibitory control — the brain’s braking system.
This braking system helps you:
When the braking system is weaker:
This is why ADHD is often described as a difficulty with self-control over time, not simply a difficulty with attention.
3. The “Time Blindness” Effect
Another important feature of ADHD is difficulty sensing and managing time.
The brain systems that help us:
… do not signal as strongly in ADHD.
This can lead to:
This is sometimes called “time blindness.”
4. Dopamine and Motivation
Dopamine is a brain chemical involved in motivation and reward.
In ADHD:
This is why people with ADHD often say:
This is a regulation difference — not laziness.
5. Emotional Regulation
ADHD also affects emotional control.
Because the same brain systems that regulate attention also regulate emotion, people with ADHD may experience:
This does not mean emotions are inappropriate — it means the volume control is more sensitive.
ADHD is best understood as:

Executive functioning is responsible for coordinating:
◆ Thinking ◆ Behaviour ◆ Emotion ◆ Organisation
◆ Motivation ◆ Planning ◆ Self-regulation in everyday life
◆ Managing behaviour over time in pursuit of goals.
These systems are heavily involved in:
◆ Motivation ◆ Reward anticipation ◆ Behavioural inhibition
◆ Working memory ◆ Sustained mental effort
In ADHD people experience difficulties not because they lack knowledge or intelligence,
but because the brain has difficulty consistently regulating:
◆ Attention ◆ Effort ◆ Motivation ◆ Emotion ◆ Behaviour across time
◆ impairments in the ability to internally guide behaviour toward
future goals, particularly when tasks lack:
◇ Immediate reward
◇ Urgency
◇ Novelty
◇ Emotional significance
◆ These processes allow individuals to:
◇ Initiate tasks
◇ Sustain attention
◇ Organise information
◇ Regulate emotions
◇ Inhibit impulses
◇ Adapt behaviour according to changing demands or future goals
◆ involving difficulties with:
◇ Inhibiting behaviour
◇ Regulating attention
◇ Managing emotions
◇ Sustaining motivation
◇ Organising behaviour across time
◆ ADHD reflects impairments in the ability to internally guide behaviour toward
future goals, particularly when tasks lack:
◇ Immediate reward
◇ Urgency
◇ Novelty
◇ Emotional significance
◆ impact:
◇ Occupational functioning
◇ Academic performance
◇ Relationships
◇ Emotional regulation
◇ Task completion
◇ Day-to-day self-management

◇ Self-Management to Time
◆ Estimating time accurately
◆ Planning ahead
◆ Prioritising tasks
◆ Meeting deadlines
◆ Maintaining awareness of future consequences
◆ Often associated with “time blindness” in ADHD
◇ Organisation and Problem Solving
◆ Planning complex tasks
◆ Sequencing information
◆ Holding multiple steps in mind
◆ Cognitive flexibility
◆ Managing competing demands
◆ Generating and implementing strategies
◇ Self-Restraint (Inhibitory Control)
◆ Impulse control
◆ Pausing before acting
◆ Filtering distractions
◆ Inhibiting emotional or behavioural responses
◆ Maintaining attention despite competing stimuli
◇ Self-Motivation
◆ Sustaining effort toward delayed rewards
◆ Initiating non-preferred tasks
◆ Persisting with mundane or repetitive activities
◆ Maintaining goal-directed behaviour without immediate reinforcement
◇ Self-Regulation of Emotions
◆ Modulating frustration, anger, anxiety, or emotional intensity
◆ Recovering from emotional activation
◆ Managing rejection sensitivity and stress responses
◆ Maintaining emotional stability during interpersonal
or cognitive demands

ADHD is best understood as a disorder of network-level dysfunction across three interconnected systems:
Disruption across these networks produces the core clinical features of ADHD:
Neurobiological integration:
ADHD does not arise from a single regional deficit. Rather, it reflects altered connectivity and communication between networks, particularly within dopaminergic pathways. Dopamine modulates signal-to-noise ratio in these circuits, influencing attention, motivation, and behavioural regulation.
When dopaminergic tone is optimal—such as during tasks that are novel, highly stimulating, or urgent—network efficiency improves. This can result in periods of intense, sustained focus and productivity (commonly described as hyperfocus).
Summary:
ADHD reflects differences in distributed brain systems responsible for:
These features arise from dynamic interactions between large-scale neural networks rather than isolated structural abnormalities.
Executive Control System
The prefrontal cortex is located at the front of the brain, particularly involving
the dorsolateral and ventrolateral regions.
Primary Functions:
The prefrontal cortex is responsible for higher-order executive processes, including:
ADHD-Related Impairments:
In Attention-Deficit/Hyperactivity Disorder, dysfunction within the prefrontal cortex
is associated with a consistent pattern of executive impairment:
Neuroimaging research consistently demonstrates reduced activation and structural
differences in prefrontal regions in individuals with ADHD, particularly within networks
responsible for attention regulation and executive control.
Problems in daily life:
What this means :
The prefrontal cortex is heavily modulated by dopamine.
In ADHD, reduced dopaminergic efficiency contributes to underactivation of this region,
resulting in diminished capacity to regulate attention, behaviour, and
This underactivation explains why ADHD is best conceptualised as a disorder of
performance (execution of behaviour in real time), rather than a disorder of knowledge.
Individuals typically understand what is required but experience difficulty translating that knowledge into consistent, goal-directed act
This system relies heavily on dopamine. In ADHD, dopamine signalling here is less consistent, which can lead to:
This helps explain why motivation in ADHD is interest-based rather than importance-based.
In ADHD, dopamine signalling is thought to be inefficient or underactive in several brain regions within frontostriatal circuits involving the:
✨prefrontal cortex ✨anterior cingulate cortex
✨ basal ganglia ✨nucleus accumbens.
This contributes to reduced signal strength within executive functioning and reward systems.
1. Dopamine and Reward Prediction
Dopamine is also involved in reward prediction error signalling — the brain’s mechanism for determining whether an activity is worth sustaining effort toward.
In ADHD, delayed or abstract rewards often fail to generate sufficient dopaminergic reinforcement.
This contributes to:
Stimulant medication improves the salience of future rewards and enhances the brain’s
capacity to sustain effort toward non-immediate goals.
ADHD has long been associated with dysregulation of the brain’s dopaminergic pathways, particularly within the mesolimbic and mesocortical circuits (Volkow et al., 2009).
Dopamine is a key neurotransmitter involved in
reward anticipation, motivation, and reinforcement learning.
In ADHD, reduced tonic dopamine levels and diminished phasic dopamine release contribute to difficulty sustaining effort for tasks that lack immediate intrinsic reward (Tripp & Wickens, 2008).
2. Motivation and Reward Processing
Individuals with ADHD often display “delay aversion” and reduced reward sensitivity—that is, a preference for immediate, smaller rewards over larger, delayed ones (Sonuga-Barke, 2005).
Neuroimaging demonstrates underactivation of the ventral striatum during reward anticipation, which correlates with reduced motivation and goal-directed persistence (Plichta & Scheres, 2014).
Consequently, ADHD is not primarily a disorder of knowing what to do but of doing what one knows, especially when external reward or novelty is low.
3. Dopamine, Executive Function, and Effort Allocation
Dopamine modulates the prefrontal cortex, influencing executive functions such as working memory, sustained attention, and effort-based decision-making.
When dopamine signalling is insufficient, tasks perceived as cognitively effortful or boring fail to activate motivational circuits (Westbrook & Braver, 2016).
This neurochemical mechanism underpins the subjective “interest-based” attention style often reported by adults with ADHD.
4. Dopamine Reward Pathway
One of the most significant differences between an ADHD brain vs. a
normal brain is the level of norepinephrine (a neurotransmitter).
Norepinephrine is synthesised from dopamine. Since the two go hand-in-hand,
experts believe that lower levels of dopamine & norepinephrine are both linked to ADHD.
An imbalance in the transmission of dopamine in the brain may be associated
with symptoms of ADHD, including inattention and impulsivity.
This disruption may also interfere with the changing how the ADHD brain
perceives reward and pleasure.
5. Two fundamental kinds of brain signalling in ADHD
Bottom-up signals
Top-down signals
The imbalance in ADHD

Many people with ADHD find that background sound actually improves focus—
and there are solid neuroscience reasons why.
The ADHD brain is under-stimulated
ADHD is linked to lower activity in brain networks responsible for attention, especially involving dopamine and norepinephrine.
This is sometimes called optimal stimulation theory.
Sound increases dopamine
Certain sounds (music, white noise, rhythmic beats) can:
ADHD brains often chase stimulation — sound supplies it safely.
It masks distracting thoughts
Without external sound:
Background noise acts like a mental noise-canceller, giving the brain something steady to latch onto.
Rhythm helps regulate attention
Steady beats or predictable sound patterns can:
This is why many people with ADHD prefer:
Lyrics can be distracting because they compete with language processing.
It reduces boredom (which shuts down focus)
ADHD attention is interest-based, not importance-based.
If a task is:
→ The brain disengages.
Sound adds stimulation so the task becomes tolerable.
Think of the ADHD brain like a car idling too low.

Rejection sensitivity (often termed Rejection Sensitivity Dysphoria, RSD) is
Core features
Why it occurs in ADHD
Clinical presentation
Key clinical distinctions
Management implications

The 7 Truths about Emotions & ADHD Video by Dr William Dobson
Managing Rejection Sensitivities in Real Time video By Dr Sharon Saline
How ADHD shapes perception, motivations & emotions Video by Dr William Dobson
Managing big emotions in ADHD Video by Dr Sharon Saline
Deficient Emotional Self Regulation in ADHD Video by Dr Barkley
Article: 3 Defining Features of ADHD That Everyone Overlooks
Article: Exaggerated Emotions: How and Why ADHD Triggers Intense Feelings
Article: Rejection Sensitivity Is Worse for Girls and Women with ADHD
Article: How ADHD Ignites RSD: Meaning & Medication Solutions
Article: New Insights Into Rejection Sensitive Dysphoria
Article: RSD Vs Bipolar Disorder
Test for Rejection Sensitivity
An ADHD guide to Emotional Dysregulation & Rejection Sensitivity Dysphoria
By Dr William Dodson Video
Rejection Sensitivity & Social Anxiety
By Dr Sharon Saline Video
How RSD presents
Internalised RSD:
Presents as sudden, intense sadness that can imitate a major mood disorder, sometimes with suicidal ideation. This rapid shift in mood is often misdiagnosed as rapid-cycling bipolar disorder or major depressive episodes.
Externalised RSD:
Manifests as instantaneous rage toward the person or situation perceived as rejecting
Can be mistaken for anger dysregulation or oppositional behaviour .
Anticipatory RSD:
Leads individuals to constantly scan for potential rejection, even when uncertain.
May resemble social phobia, though the core fear is different
Social Anxiety Vs Rejection Sensitivity:
Social phobia: fear of public humiliation or negative scrutiny.
RSD: fear of losing love, approval, or respect.
Subjective Experience:
People often struggle to put RSD into words. They describe it as Intense, Awful, Terrible, Overwhelming
The emotional reaction is consistently tied to a perceived or real loss of approval, love, or respect.
Rejection Sensitivity Dysphoria (RSD) in Attention-Deficit/Hyperactivity Disorder (ADHD)
reflects altered neural processing of :
social threat, emotional salience, and regulation, rather than a discrete diagnostic entity.
It arises from functional and connectivity differences across fronto-limbic, salience, and reward networks.
1. Amygdala: Heightened Threat Detection
The amygdala plays a central role in detecting threat and assigning emotional salience, particularly to social cues such as criticism, exclusion, or perceived disapproval.
Importantly, this response is fast and reflexive, occurring before higher-order cognitive appraisal.
2. Prefrontal Cortex (PFC): Impaired Top-Down Regulation
The prefrontal cortex—particularly the dorsolateral (dlPFC) and ventromedial (vmPFC) regions—modulates emotional responses generated by the limbic system.
In ADHD:
This explains why RSD is often described as overwhelming, uncontrollable, and physically painful.
3. Anterior Cingulate Cortex (ACC): Pain and Social Error Processing
The anterior cingulate cortex integrates emotional pain, cognitive conflict, and social evaluation.
This shared circuitry helps explain why rejection in RSD is often described as visceral rather than merely
4. Dopaminergic Reward Pathways: Salience Without Stability
ADHD is associated with dopaminergic dysregulation in fronto-striatal circuits, including the nucleus accumbens.
As a result, rejection signals carry disproportionate motivational and emotional weight.
5. Default Mode Network (DMN): Internalisation and Rumination
The default mode network, active during self-referential thought, shows atypical regulation in ADHD.
This sustains RSD responses well beyond the initial interpersonal cue.
Integrated Neurobiological Model of RSD in ADHD
RSD reflects the convergence of:
Together, these systems produce rapid, intense, and enduring emotional responses
to perceived rejection, often without conscious control.
Prenatal and Early Life Risk Factors of ADHD: What Research Says — and What Parents Can Do.
Additude Article September 2025
For most people with ADHD, many genetic and environmental risk factors accumulate to cause the disorder (Faraone et al., 2015).
The environmental risks for ADHD exert their effects very early in life, during the fetal or early postnatal period. In rare cases, however, ADHD-like symptoms can be caused by extreme deprivation early in life (Kennedy et al., 2016), a single genetic abnormality (Faraone and Larsson, 2018), or traumatic brain injury early in life (Stojanovski et al., 2019).
These findings are helpful to understand the causes of ADHD but are not useful for diagnosing the disorder.
The associations between aspects of the environment and the onset of ADHD have attained a very high level of evidential support. Some have strong evidence for a causal role but, for most, the possibility remains that these associations are due to correlated genetic and environmental effects.
For this reason, we refer to features of the pre- and post-natal environments that increase risk for ADHD as correlates, rather than causes.
The genetic and environmental risks described below are not necessarily specific to ADHD.
ADHD can also be the result of rare single gene defects (Faraone and Larsson, 2018) or abnormalities of the chromosomes (Cederlof et al., 2014). When the DNA of 8000+ children with autism spectrum disorder (ASD) and/or ADHD and 5000 controls was analyzed, those with ASD and those with ADHD had an increased rate of rare genetic mutations compared with controls (Satterstrom et al., 2019).
A review of 37 twin studies from the United States, Europe, Scandinavia, and Australia found that genes and their interaction with the environment must play a substantial role in causing ADHD (Faraone and Larsson, 2018; Larsson et al., 2014a; Pettersson et al., 2019).
In a genomewide study, an international team analysed DNA from over 20,000 people with ADHD and over 35,000 without ADHD from the United States, Europe, Scandinavia, China, and Australia. They identified many genetic risk variants, each having a small effect on the risk for the disorder (Demontis et al., 2019).
This study confirmed a polygenic cause for most cases of ADHD, meaning that many genetic variants, each having a very small effect, combine to increase risk for the disorder. The polygenic risk for ADHD is associated with general psychopathology (Brikell et al., 2020) and several psychiatric disorders (Lee et al., 2019a,b
Family, twin, and DNA studies show that genetic and environmental influences are partially shared between ADHD and many other psychiatric disorders (e.g. schizophrenia, depression, bipolar disorder, autism spectrum disorder, conduct disorder, eating disorders, and substance usedisorders) and with somatic disorders (e.g. migraine and obesity) (Demontis et al., 2019) (Faraone and Larsson, 2018) (Ghirardi et al., 2018) (Lee et al., 2019a,b) (Lee et al., 2013) (Anttila et al., 2018; Tylee et al., 2018) (van Hulzen et al., 2017) (Vink and Schellekens, 2018) (Brikell et al., 2018) (Chen et al., 2019a) (Yao et al., 2019).
However, there is also a unique genetic risk for ADHD.
Evidence of shared genetic and environmental risks among disorders suggest that these disorders also share a pathophysiology in the biological pathways that dysregulate neurodevelopment and create brain variations leading to disorder onset.
Very large studies of families suggest that ADHD shares genetic or familial causes with autoimmune diseases (Li et al., 2019), hypospadias (Butwicka et al., 2015), and intellectual disability (Faraone and Larsson, 2018).

Understanding ADHD Burnout: Causes, Symptoms, and Coping Strategies
Living with ADHD (Attention Deficit Hyperactivity Disorder) means navigating a brain that works differently. While it comes with strengths—like creativity, energy, and unique problem-solving—it also brings daily challenges around focus, organization, and emotional regulation. Add in the constant pressure to keep up in a fast-paced world, and many people with ADHD eventually hit a wall: ADHD burnout.
This type of burnout is often misunderstood, but it’s a very real and overwhelming experience.
Let’s dive into what it is, why it happens, and how to recover when you feel completely drained.
What Is ADHD Burnout?
ADHD burnout is a state of mental, emotional, and physical exhaustion. It happens when someone with ADHD pushes themselves too hard—often trying to meet external expectations—while also managing the everyday demands that ADHD makes harder.
Some of the key drivers include:
Over time, this pressure adds up, leaving you feeling depleted, stuck, and hopeless.
Causes of ADHD Burnout
There isn’t just one reason ADHD burnout happens—it’s usually a mix of factors. Here are some of the most common:
Cognitive Overload
Because ADHD impacts executive function, everyday tasks like planning, prioritizing, or meeting deadlines require extra effort. That constant mental strain leads to fatigue.
Hyperfocus (and the Crash After)
When someone with ADHD locks into a task, they can work for hours straight—forgetting meals, breaks, or rest. Eventually, the body and brain crash, creating burnout.
Inconsistent Motivation
Tasks that feel boring can be almost impossible to start, while interesting ones may lead to overwork. That rollercoaster creates stress and imbalance.
Social and Emotional Stress
ADHD often comes with feeling misunderstood, judged, or “not enough.” Social interactions can feel draining, and emotional stress compounds the burnout cycle.
Masking
Trying to hide ADHD traits in professional or social settings takes huge effort. Over time, that performance is exhausting.
Sleep Struggles
Many people with ADHD wrestle with insomnia, racing thoughts, or irregular sleep patterns. Poor rest makes recovery from burnout even harder.
Signs and Symptoms of ADHD Burnout
ADHD burnout can look different for everyone, but some common signs include:
Coping Strategies: How to Recover from ADHD Burnout
The good news is: recovery is possible. It takes patience, self-compassion, and strategies that work with your ADHD brain—not against it. Here are some steps to help you reset:
1. Recognize the Signs Early
Burnout sneaks up on you. Noticing the red flags (irritability, brain fog, emotional fatigue) gives you a chance to pause before things get worse.
2. Lower the Bar
Set realistic expectations. Perfectionism and impossible standards feed burnout—aim for “good enough” instead of “perfect.”
3. Use ADHD-Friendly Tools
4. Build in Breaks
Schedule downtime the way you would schedule a meeting. Your brain needs recovery time.
5. Practice Self-Compassion
ADHD isn’t about laziness or weakness—it’s neurological. Speak to yourself kindly and give yourself permission to rest.


All explanations and text are taken from the extensive work of Dr William Dobson who devised the term after extensive research.
The 7 Truths about Emotions & ADHD Video by Dr William Dobson
Managing Rejection Sensitivities in Real Time video By Dr Sharon Saline
How ADHD shapes perception, motivations & emotions Video by Dr William Dobson
Managing big emotions in ADHD Video by Dr Sharon Saline
Deficient Emotional Self Regulation in ADHD Video by Dr Barkley
Article: 3 Defining Features of ADHD That Everyone Overlooks
Article: Exaggerated Emotions: How and Why ADHD Triggers Intense Feelings
Article: Rejection Sensitivity Is Worse for Girls and Women with ADHD
Article: How ADHD Ignites RSD: Meaning & Medication Solutions
Article: New Insights Into Rejection Sensitive Dysphoria
Article: RSD Vs Bipolar Disorder
Rejection sensitive dysphoria (RSD) is an intense vulnerability to the perceptio — not
necessarily the reality — of being rejected, teased, or criticised by important people
in your life. RSD causes extreme emotional pain that may also be triggered by a sense
of failure, or falling short — failing to meet either your own high standards or others’
expectation.
Dysphoria is the Greek word meaning unbearable; its use emphasizes the severe physical and
emotional pain suffered by people with RSD when they encounter real or perceived
rejection, criticism, or teasing.
The response is well beyond all proportion to the nature of the event that triggered it.
Rejection sensitive dysphoria is not a formal diagnosis, but rather one of the most
common and disruptive manifestations of emotional dysregulation—a common but
under-researched and oft-misunderstood symptom of ADHD, particularly in adults.
RSD is a brain-based symptom that is likely an innate feature of ADHD.
Often, this intense emotional reaction is hidden from other people. People
experiencing it don’t want to talk about it because of the shame they feel over their lack
control, or because they don’t want people to know about this intense vulnerability.
Test for Rejection Sensitivity
An ADHD guide to Emotional Dysregulation & Rejection Sensitivity Dysphoria
By Dr William Dodson Video
Rejection Sensitivity & Social Anxiety
By Dr Sharon Saline Video
How RSD presents
Internalised RSD:
Presents as sudden, intense sadness that can imitate a major mood disorder, sometimes with suicidal ideation. This rapid shift in mood is often misdiagnosed as rapid-cycling bipolar disorder or major depressive episodes.
Externalised RSD:
Manifests as instantaneous rage toward the person or situation perceived as rejecting
Can be mistaken for anger dysregulation or oppositional behavior.
Anticipatory RSD:
Leads individuals to constantly scan for potential rejection, even when uncertain.
May resemble social phobia, though the core fear is different
Social Anxiety Vs Rejection Sensitivity:
Social phobia: fear of public humiliation or negative scrutiny.
RSD: fear of losing love, approval, or respect.
Subjective Experience:
People often struggle to put RSD into words. They describe it as Intense, Awful, Terrible, Overwhelming
The emotional reaction is consistently tied to a perceived or real loss of approval, love, or respect.

Article: Lifetime co-occurring psychiatric disorders in newly diagnosed adults with
ADHD and Autism Spectrum Disorder. (2020)
Video : Anxiety & ADHD: How they are related By Dr Barkley
Perfectionism & ADHD & Anxiety By Dr Sharon Saline video
Emotional Dysregulation & Rejection Sensitivity Dysphoria
By Dr William Dodson Video (60 mins)
ADHD & Social Anxiety
By Dr Sharon Saline
Article: Females with ADHD: a lifespan approach in girls and women.
BMC Psychiatry (2020)
Article: Annual Research Review: ADHD in girls and women: underrepresentation,
longitudinal processes, and key directions.
Stephen P. Hinshaw. Journal of Child Psychology and Psychiatry (2021)
Article: Adverse experiences of women with undiagnosed ADHD and the invaluable role of diagnosis.
Holden, E., Kobayashi-Wood, H. Sci Rep 15, 20945 (2025).
Article: ADHD and Sex Hormones in Females: A Systematic Review .
J Atten Disord. 2025 Apr 18;29(9):706–723.
Article : Exploring Female Students’ Experiences of ADHD and its Impact on Social, Academic, and Psychological Functioning.
J Atten Disord. 2023 Aug;27(10):1129-1155.
ADHD in women and girls is frequently overlooked when anxiety or depression is present, because ADHD symptoms—especially inattention, restlessness, and emotional lability—are misattributed to the mood or anxiety disorder. Women with ADHD have a high likelihood of also experiencing anxiety disorders (generalised anxiety disorder, social anxiety, panic disorder, or phobias).
Studies estimate that 25–50% of women with ADHD meet criteria for at least one anxiety disorder at some point. Undiagnosed and untreated ADHD can frequently lead to a secondary presentation of general anxiety , triggered by the consequences , in home and work life, of challenges with procrastination, distractibility, time management, planning and setting and following through on priorities.
Frequently Woman are often treated first for anxiety and/or depression, delaying recognition of the under diagnosed ADHD.

This section is included to explain where misdiagnosis can occur. Misdiagnosis of Bipolar Affective Disorder when it may be ADHD.
At RMC we have an in depth understanding of the difference in both presentations.
Dr Lewis has a specialisation in the psychiatric treatment of combined ADHD and Bipolar Affective Disorder, and has 27 years of experience of psychiatric treatment of Bipolar 1 disorder and Bipolar 2 disorder.
An ADHD guide to Emotional Dysregulation & Rejection Sensitivity Dysphoria
By Dr William Dodson Video
https://youtu.be/yipQQk2iALQ?si=6vVmtc_8JSgDtPFq
https://youtu.be/52GqJJdosxQ?si=wUQPUguNnzlH1ipD
https://youtu.be/ibPRV_DocmQ?si=-bJFN1eFVNl2dj-Z
Bipolar was formerly called manic-depressive illness or manic depression.
Bipolar Affective Disorder & ADHD share 14 features in common.Two studies, the STAR*D program
and the STEP-BD program, both found a tremendous overlap between the disorders.
For clients with bipolar disorder, there is a 40% chance of having ADHD as well.
ADHD:
With adult ADHD, you see a very di!erent pattern; the moods of an individual with
ADHD are clearly triggered. The ADHD symptoms of rejection sensitive dysphoria, for example,
is triggered by the perception that a person has been rejected, teased, or criticised.
An observer might not be able to point out the trigger, but the individual
with ADHD can say, “When my mood shifts, I can always see a trigger.
My mood matches my perception of the trigger.” In technical terms, ADHD moods are “congruent.”
Mood changes are instantaneous and intense in individuals with ADHD, much
more so than in a neuro-typical person.
ADHD moods rarely persist for more than a few hours. It is extremely
rare for them to last two weeks. Typically, the mood can be altered by
the person with ADHD finding a new interest or occupation that
captures their interest and distracts them from the intense emotion.
Bipolar Mood Disorder:
Unlike ADHD, bipolar is a classic mood disorder that has a life of its own separate from
the events of a person’s life, outside of the person’s conscious will and control.
Bipolar moods aren’t necessarily triggered by something; they just come and they stay.
Usually, the onset is very gradual over a period of weeks to months.
To meet the bipolar definition, the mood must be continuously present for at least
two weeks and then its resolution must be gradual over a period of weeks to months.
Depression and anxiety are often the most visible coexisting conditions experienced by women who have ADHD. Both conditions can be present as separate disorders or as the result of struggling with undiagnosed or poorly treated ADHD for a very long time.
The two conditions frequently prompt women to seek medical and mental health care and can lead to a diagnosis of ADHD.
When treating a woman or girl who has ADHD and a co-occurring condition, the clinician or treatment specialist needs to address the condition causing the most difficulty at that moment, especially conditions that can be life-threatening if untreated.
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